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Rheumatology 2000; 39: 692-695
© 2000 British Society for Rheumatology

Editorials

Reflex sympathetic dystrophy

A. T. Marshall and A. J. Crisp1

Department of Rheumatology, Norfolk and Norwich NHS Trust, Brunswick Road, Norwich NR1 3SR,
1 Department of Rheumatology, Addenbrooke's NHS Trust, Hills Road, Cambridge CB2 2QQ, UK

The first 150 words of the full text of this article appear below.

Reflex sympathetic dystrophy (RSD) is a condition characterized by localized or diffuse pain, usually with associated swelling, trophic changes and vasomotor disturbance [1]. Allodynia, hyperhydrosis, and nail or hair growth changes may also occur. Motor abnormalities have been reported [2], in particular, tremor, involuntary movement and muscle spasm. Contractures may occur in the later stages. Involvement may be either unilateral or bilateral. Most commonly recognized is peripheral disease [3], although RSD may affect any region of the trunk or limbs. There is often a history of trauma, occasionally of such low significance that it may be overlooked by the patient. Symptoms may occur up to 6 months after injury [4]. Other triggering factors have been reported. Several drugs have been implicated, for example, phenobarbitone, phenytoin, isoniazid [5], and the immunosuppressive agents cyclosporin [6] and tacrolimus [7], . . . [Full Text of this Article]


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