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Rheumatology 2001; 40: 363-366
© 2001 British Society for Rheumatology
Editorial |
The aetiology of psoriatic arthritis
Central Manchester Healthcare NHS Trust and University of Manchester and
1 Arthritis Research Campaign Epidemiology Unit, University of Manchester, Stopford Building, Oxford Road, Manchester M13 9PT, UK
| The first 150 words of the full text of this article appear below. |
There is little question that psoriatic arthropathy (PsA) is a poorly studied entity. At the recent 64th American College of Rheumatology meeting, abstracts relating to PsA were out numbered five to one by studies on systemic lupus erythematosus SLE, a condition of similar prevalence. The concept of PsA is not universally accepted, and even among those who accept the concept there is no internationally agreed definition or diagnostic criteria. For example, while Moll and Wright's [1] original definition described PsA as an inflammatory arthritis associated with psoriasis and usually negative for rheumatoid factor, some groups have excluded patients who are positive for rheumatoid factor [2].
It is accepted that psoriasis and inflammatory arthritis coexist more frequently than would be expected by the coincidence of two common conditions, i.e. psoriasis and rheumatoid arthritis (RA) [3, 4]. The explanation for this increased association has been
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