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Rheumatology Advance Access originally published online on November 18, 2006
Rheumatology 2007 46(4):559-560; doi:10.1093/rheumatology/kel377
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© The Author 2006. Published by Oxford University Press on behalf of the British Society for Rheumatology. All rights reserved. For Permissions, please email: journals.permissions@oxfordjournals.org

EDITORIALS

18F-Flourodeoxyglucose positron emission tomography in polymyalgia rheumatica: novel insight into complex pathogenesis but questionable use in predicting relapses

C. Dejaco, C. Duftner, E. Wipfler and M. Schirmer

General Hospital of the Elisabethinen, Voelkermarkterstrasse 15-19, A-9020 Klagenfurt, Austria.

Correspondence to: M. Schirmer MD. E-mail: michael.schirmer@ekh.at

The first 10% of the full text of this article appears below.

Polymyalgia rheumatica (PMR) is the most common inflammatory rheumatic disease in the elderly and is characterized by headache, bilateral pain and stiffness in the neck, shoulders and pelvic girdle [1, 2]. PMR occurs isolated or concomitantly with giant-cell arteritis (GCA), a chronic vasculitis of large- and medium-sized vessels that is associated with severe complications including blindness, aortic stenosis, dissections and aneurysms [3]. Although higher doses of steroids are required to suppress inflammation in GCA, patients with isolated PMR usually show rapid response to low doses of corticosteroids [3]. Most patients with isolated PMR then withdraw steroids after 1–2 yrs [3], but up to one-third of patients have a more chronic, relapsing course requiring low doses of corticosteroids for . . . [Full Text of this Article]


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