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Rheumatology Advance Access originally published online on July 10, 2007
Rheumatology 2007 46(9):1385-1387; doi:10.1093/rheumatology/kem163
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© The Author 2007. Published by Oxford University Press on behalf of the British Society for Rheumatology. All rights reserved. For Permissions, please email: journals.permissions@oxfordjournals.org

EDITORIALS

The Rheumatoid nodule: peripheral or central to rheumatoid arthritis?

J. Highton, P. A. Hessian1 and L. Stamp2

Medicine, Medical and Surgical Sciences, University of Otago, Dunedin School of Medicine,1Physiology Department, University of Otago, Otago School of Medical Sciences, PO Box 913, Dunedin and 2Senior Lecturer and Consultant Rheumatologist, University of Otago, Christchurch School of Medicine, Christchurch Hospital, Christchurch, New Zealand

Correspondence to: Prof. John Highton. E-mail: john.highton@stonebow.otago.ac.nz

The first 150 words of the full text of this article appear below.

When asked as a medical student how to treat a patient with rheumatoid arthritis (RA) who was not doing well the immediate answer was gold. The question was not hard since a flick through a short textbook prior to the clinic had not indicated many alternatives. How should the modern medical student answer this question? What might be the best of many treatment options for an individual patient? At least in the case of the newer biological agents their use is based on some understanding of the basic processes involved in the pathogenesis of RA. Can we therefore apply this understanding in making a choice of treatment? Unfortunately any current review of the pathogenesis of RA reveals a situation at least as complex as the therapeutic choices. This has lead Cornelia Weyand [1] to suggest that one way forward would be to develop a string theory of autoimmunity. . . . [Full Text of this Article]


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