Skip Navigation


Rheumatology Advance Access originally published online on November 22, 2007
Rheumatology 2008 47(1):103-104; doi:10.1093/rheumatology/kem259
This Article
Right arrow Full Text
Right arrow Full Text (PDF)
Right arrow All Versions of this Article:
47/1/103    most recent
kem259v1
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrowRequest Permissions
Right arrow Disclaimer
Google Scholar
Right arrow Articles by Gubbels Bupp, M. R.
Right arrow Articles by Peng, S. L.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Gubbels Bupp, M. R.
Right arrow Articles by Peng, S. L.
Related Collections
Right arrow Experimental Arthritis
Right arrow Immunogenetics
Social Bookmarking
Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

© The Author 2007. Published by Oxford University Press on behalf of the British Society for Rheumatology. All rights reserved. For Permissions, please email: journals.permissions@oxfordjournals.org

LETTERS TO THE EDITOR

The candidate lupus susceptibility gene Ifi202a is largely dispensable for B-cell function

M. R. Gubbels Bupp, M. Li, A. Pashine, D. Aud and S. L. Peng

Roche Palo Alto, Palo Alto, CA, USA

Correspondence to: S. L. Peng, 3431 Hillview Ave., M/S A2-259, Palo Alto, CA 94304, USA. E-mail: stanford.peng@roche.com

The first 10% of the full text of this article appears below.

SIR, The transcriptional modulator Ifi202 has been implicated in the pathogenesis of lupus via genetic studies in the vicinity of the Nba2 locus [1]. It is a member of the interferon-inducible Ifi200 cluster of genes located on mouse chromosome 1, which bears homology to a cluster of three human chromosome 1 genes (MNDA, IFI 16 and AIM2 [2]), all of which share a largely conserved ~200 amino acid domain and have been implicated in multiple cellular processes, including cellular proliferation and apoptosis, by . . . [Full Text of this Article]


Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?


This article has been cited by other articles:


Home page
 Rheumatology (Oxford)Home page
D. Choubey
Comment on: The candidate lupus susceptibility gene Ifi202a is largely dispensable for B-cell function
Rheumatology, April 1, 2008; 47(4): 558 - 559.
[Full Text] [PDF]