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Rheumatology Advance Access originally published online on April 27, 2009
Rheumatology 2009 48(7):855-857; doi:10.1093/rheumatology/kep094
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© The Author 2009. Published by Oxford University Press on behalf of the British Society for Rheumatology. All rights reserved. For Permissions, please email: journals.permissions@oxfordjournals.org

Monozygotic twins with distinct forms of idiopathic inflammatory myositis

Vidya Limaye1,2, Jenny Walker3, Michael Ahern4, Peter Bardy5, Sally Cox6, Peter Roberts-Thomson7, Sue Lester8 and Peter Blumbergs9

1Department of Rheumatology, Royal Adelaide Hospital, North Tce, 2Department of Medicine, University of Adelaide, 3Department of Rheumatology, Flinders University, Adelaide, 4Department of Rheumatology, Repatriation General Hospital, Daw Park,5Red Cross Blood Service, 6Department of Rheumatology, Flinders Medical Centre, Bedford Park, 7Department of Immunology, SA Pathology, 8Arthritis Research Laboratory, Hanson Institute, 9Neuropathology Laboratory, Hanson Institute, Institute of Medical and Veterinary Science, Adelaide, South Australia, Australia

Correspondence to: Vidya Limaye, Department of Rheumatology, Royal Adelaide Hospital, North Tce, Adelaide SA 5000, South Australia, Australia. E-mail: vidya.limaye@health.sa.gov.au

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SIR, A 56-year-old female (Patient A) presented with leg weakness of 4 years duration. Examination showed weakness of hip flexors and extensors, knee flexors and extensors and weakness of finger flexors confirmed with dynomometry. Manual muscle testing showed that power was 3+/5 in finger flexors, 4/5 in forearm pronators and supinators and 4/5 in hip rotation. Investigations revealed: creatine kinase (CK), 605 U/l [normal range (NR)<150]; lactate dehydrogenase (LDH), 382 U/l (NR 115–200); CRP, 1 mg/l (NR<6); RF 48 IU/l (NR<40); negative antibodies to ENAs; and negative ANA . . . [Full Text of this Article]


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