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Rheumatology 2000; 39: 447-448
© 2000 British Society for Rheumatology

Letters to the Editor

A case of hypopituitarism associated with antiphospholipid syndrome

K. IKEDA, M. TAKAHAMA, M. MATSUSHITA, S. ANDO, I. SEKIGAWA, N. IIDA and H. HASHIMOTO1

Department of Medicine, Juntendo University, Izu-Nagaoka Hospital, 1129 Nagaoka, Izu-Nagaoka-cho, Tagata-gun, Shizuoka 410–2295,
1 Department of Internal Medicine and Rheumatology, Juntendo University School of Medicine, 2-1-1 Hongo, Bunkyo-ku, Tokyo 113, Japan

SIR, The major clinical features of antiphospholipid syndrome (APS) are thromboses, thrombocytopenia, and recurrent fetal demise. Recent reports have indicated that antiphospholipid antibodies (aPL) are also related to other clinical manifestations, including cardiac valve lesions and haemolytic anaemia with or without underlining autoimmune diseases such as systemic lupus erythematosus (SLE), although the precise mechanism of aPL on the induction of these features is still unclear [1]. We recently encountered a case of multiple hormonal deficiencies in the pituitary gland associated . . . [Full Text of this Article]

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I. Sekigawa, K. Ikeda, J. Suzuki, M. Nawata, S. Lee, N. Iida, and H. Hashimoto
Novel clinical manifestations associated with antiphospholipid antibodies
Rheumatology, December 1, 2000; 39(12): 1437 - 1439.
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