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Rheumatology Advance Access originally published online on September 27, 2005
Rheumatology 2005 44(11):1456-1457; doi:10.1093/rheumatology/kei120
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© The Author 2005. Published by Oxford University Press on behalf of the British Society for Rheumatology. All rights reserved. For Permissions, please email: journals.permissions@oxfordjournals.org

LETTER TO THE EDITOR

Polymorphisms of the toll-like receptor 9 (TLR9) gene with systemic lupus erythematosus in Chinese

M. W. Ng, C. S. Lau1, T. M. Chan1, W. H. S. Wong and Y. L. Lau

Department of Paediatrics and Adolescent Medicine and 1 Department of Medicine, Queen Mary Hospital, University of Hong Kong, Pokfulam and Genome Research Centre, University of Hong Kong, Hong Kong SAR, China

Correspondence to: Y. L. Lau, Department of Paediatrics and Adolescent Medicine, Queen Mary Hospital, University of Hong Kong, Pokfulam, Hong Kong SAR, China.E-mail: lauylung@hkucc.hku.hk

The first 10% of the full text of this article appears below.

SIR, Systemic lupus erythematosus (SLE) patients and murine lupus models of SLE are characterized by the presence of autoantibodies that recognize DNA and nucleosomes. It has been shown that interaction of bacterial CpG DNA with toll-like receptor 9 (TLR9) expressed on B cells can stimulate production of antibodies [1]. In the lupus mouse model and SLE patients, DNA fragments isolated from plasma are hypomethylated and they may mimic microbial DNA and . . . [Full Text of this Article]


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