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Rheumatology Advance Access originally published online on December 14, 2004
Rheumatology 2005 44(4):557-558; doi:10.1093/rheumatology/keh519
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Rheumatology Vol. 44 No. 4 © British Society for Rheumatology 2004; all rights reserved

LETTER TO THE EDITOR

Corynebacterium striatum endocarditis masquerading as connective tissue disorders

B. Stoddart, J. A. T. Sandoe and M. Denton

Medical Microbiology, Leeds Teaching Hospitals NHS Trust, Leeds, UK

Correspondence to: B. Stoddart. E-mail: bethanstoddart@hotmail.com

The first 10% of the full text of this article appears below.

SIR, Stimulation of both humoral and cellular immunity occurs in bacterial endocarditis. The constant intravascular antigenic challenge results in development of several classes of circulating antibody [1]. Deposition of immune complexes in subendothelial tissues, with associated acute inflammatory injury, leads to renal impairment and arthritis in a similar manner to some vasculitides. Treatment with antibiotics leads to a rapid fall in immune complex concentration and a clinical improvement. Immunosuppressive agents transiently have the same effect.

Anti-neutrophil cytoplasmic antibodies (ANCA) . . . [Full Text of this Article]


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