Skip Navigation


Rheumatology Advance Access originally published online on March 15, 2005
Rheumatology 2005 44(6):820-821; doi:10.1093/rheumatology/keh606
This Article
Right arrow Full Text Freely available
Right arrow FREE Full Text (PDF) Freely available
Right arrow All Versions of this Article:
44/6/820    most recent
keh606v1
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in ISI Web of Science
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrow Search for citing articles in:
ISI Web of Science (4)
Right arrowRequest Permissions
Right arrow Disclaimer
Google Scholar
Right arrow Articles by Butt, C.
Right arrow Articles by Rahman, P.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Butt, C.
Right arrow Articles by Rahman, P.
Related Collections
Right arrow Spondylarthropathies
Right arrow Immunogenetics
Social Bookmarking
Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

© The Author 2005. Published by Oxford University Press on behalf of the British Society for Rheumatology. All rights reserved. For Permissions, please email: journals.permissions@oupjournals.org

LETTER TO THE EDITOR

Lack of association of SLC22A4, SLC22A5, SLC9A3R1 and RUNX1 variants in psoriatic arthritis

C. Butt, S. Sun1, C. Greenwood1, D. Gladman2 and P. Rahman

Memorial University of Newfoundland, 1 Genetics and Genomic Biology, Hospital for Sick Children, Department of Public Health Sciences, University of Toronto, 2 Toronto Western Hospital, University of Toronto, Canada

Correspondence to: P. Rahman, St Clare's Mercy Hospital, 1 South–154 LeMarchant Rd, St John's, Newfoundland, Canada A1C–5B8. E-mail: prahman@mun.ca

The first 10% of the full text of this article appears below.

SIR, Psoriatic arthritis (PsA) is an inflammatory arthritis associated with various extra-articular features, including psoriasis (which is seen in the majority of subjects with PsA) and, occasionally, inflammatory bowel disease. Recently, there have been major advances in the genetics of psoriasis (SLC9A3R1) [1], rheumatoid arthritis (SLC22A4 and RUNX1) [2], and Crohn's disease (a haplotype of SNPs in SLC22A4 and SLC22A5) [3]. As SLC22A4, SLC22A5, SLC9A3R1 and RUNX1 have a proposed . . . [Full Text of this Article]


Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?


This article has been cited by other articles:


Home page
 Rheumatology (Oxford)Home page
J. Wesoly, R. E. M. Toes, P. E. Slagboom, and T. W. J. Huizinga
RUNX1 intronic SNP is not associated with rheumatoid arthritis susceptibility in Dutch Caucasians
Rheumatology, September 1, 2005; 44(9): 1196 - 1196.
[Full Text] [PDF]