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Rheumatology Advance Access originally published online on August 25, 2006
Rheumatology 2006 45(11):1442-1444; doi:10.1093/rheumatology/kel272
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© The Author 2006. Published by Oxford University Press on behalf of the British Society for Rheumatology. All rights reserved. For Permissions, please email: journals.permissions@oxfordjournals.org

LETTERS TO THE EDITOR

Study of the role of a functional polymorphism of MHC2TA in rheumatoid arthritis in three ethnically different populations

G. Orozco1,*, G. Robledo1,*, M. V. P. Linga Reddy2, A. García3, D. Pascual-Salcedo4, A. Balsa4, M. A. González-Gay5, A. Eimon6, S. Paira7, H. R. Scherbarth8, B. A. Pons-Estel9, I. F. Petersson10,11, M. Alarcón-Riquelme2 and J. Martín1

1Instituto de Parasitología y Biomedicina López Neyra, Granada, Spain, 2Department of Genetics and Pathology, Rudbeck Laboratory, Uppsala University, Uppsala, Sweden, 3Servicio de Reumatología, Hospital Virgen de las Nieves, Granada, Spain, 4Servicio de Reumatologia e Inmunología, Hospital La Paz, Madrid, Spain, 5Servicio de Reumatología, Hospital Xeral-Calde, Lugo, Spain, 6Centro de Educación Médica e Investigaciones Clínicas (CEMIC), Buenos Aires, Argentina, 7Hospital José M. Cullen, Santa Fe, Argentina, 8Servicio de Reumatología, Hospital Interzonal General de Agudos "Dr. Oscar Alende", Mar del Plata, Argentina, 9Sanatorio Parque, Rosario, Argentina, 10Spenshult Hospital for Rheumatic Diseases, Halmstad, Sweden and 11for the BARFOT-study group, Sweden

Correspondence to: J. Martín, MD, PhD Instituto de Parasitología y Biomedicina "López Neyra", CSIC Parque Tecnológico Ciencias de la Salud. Avenida del Conocimiento s/n. 18100 Armilla, Granada, Spain. E-mail: martin@ipb.csic.es

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SIR, Rheumatoid arthritis (RA) is a chronic complex inflammatory disease that is thought to have an autoimmune origin. Although the precise aetiology of RA is unknown, a strong genetic component is well-established. The strongest genetic association with RA has been found for particular alleles of HLA-DRB1. Furthermore, it has been reported that the differential expression of HLA class II genes is associated with both susceptibility and progression of RA [1]. Also, it has been recently reported about an association between the more severe forms of RA (extra-articular RA) and the presence of the HLA-DRB1*04 allele as well as between the HLA C3 . . . [Full Text of this Article]


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