Rheumatology Advance Access originally published online on September 23, 2006
Rheumatology 2007 46(1):178-179; doi:10.1093/rheumatology/kel322
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© The Author 2006. Published by Oxford University Press on behalf of the British Society for Rheumatology. All rights reserved. For Permissions, please email: journals.permissions@oxfordjournals.org
Unstable diabetes in a patient receiving anti-TNF-
for rheumatoid arthritis
Royal Hallamshire Hospital, Glossop Road, Sheffield, and 1Chesterfield Royal Hospital Foundation Trust, Chesterfield, UK.
Correspondence to: John Boulton, Specialist Registrar, Department of Rheumatology, Royal Hallamshire Hospital, Sheffield. E-mail: jgboulton@btinternet.com
| The first 10% of the full text of this article appears below. |
SIR, Tumour necrosis factor-
(TNF-
) is a cytokine well-recognized as having a significant role in the inflammatory process. Recent advances have led to the production of drugs that inhibit the action of TNF-
, producing significant improvement in the control of rheumatic diseases [1]. TNF-
may also play a role in other physiological
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