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Rheumatology Advance Access originally published online on May 4, 2007
Rheumatology 2007 46(8):1375-1376; doi:10.1093/rheumatology/kem094
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© The Author 2007. Published by Oxford University Press on behalf of the British Society for Rheumatology. All rights reserved. For Permissions, please email: journals.permissions@oxfordjournals.org

Meningeal involvement in apparently ANCA-negative Wegener's granulomatosis: a role for PR3 capture-ELISA?

P. Greco1, A. Palmisano1, A. Vaglio1, U. Scoditti2, M. Y. Antonelli2, G. Crisi3, R. A. Sinico4 and C. Buzio1

1Department of Clinical Medicine, Nephrology and Health Science, 2Department of Neuroscience, Section of Neurology, 3Section of Neuroradiology, University of Parma, Parma, Italy, and 4Clinical Immunology Unit, S. Carlo Borromeo Hospital, Milano, Italy

Correspondence to: Prof Carlo Buzio, Dipartimento di Clinica Medica, Nefrologia e Scienze della Prevenzione, Università degli Studi di Parma, Via Gramsci 14, 43100 Parma, Italy. E-mail: carlo.buzio@unipr.it

The first 10% of the full text of this article appears below.

SIR, Central nervous system (CNS) involvement is rare in Wegener's granulomatosis (WG): the spectrum of CNS manifestations includes cerebrovascular events, seizures and cranial nerve abnormalities [1, 2]. Meningitis is exceedingly rare, and its diagnosis often challenging [1–3].

A 57-yr-old man was admitted to hospital because of untreatable headache, photophobia, neck stiffness and paranasal sinus pain. Eight years earlier. WG had been diagnosed because of inflammatory involvement of the upper (sinusitis, anosmia) and lower (pulmonary nodules, histologically showing granulomas with giant cells and necrotizing vasculitis) . . . [Full Text of this Article]


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