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Rheumatology Advance Access originally published online on October 23, 2007
Rheumatology 2008 47(1):102-103; doi:10.1093/rheumatology/kem242
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© The Author 2007. Published by Oxford University Press on behalf of the British Society for Rheumatology. All rights reserved. For Permissions, please email: journals.permissions@oxfordjournals.org

LETTERS TO THE EDITOR

Study of two polymorphisms of the MHC2TA gene with systemic lupus erythematosus

E. Sánchez, J. M. Sabio1, J. Jiménez-Alonso1, J. L. Callejas2, M. Camps3, E. de Ramón3, R. García-Portales4, M. de Haro4, N. Ortego-Centeno2, M. A. López-Nevot5 and J. Martín

Consejo Superior de Investigaciones Científicas, CSIC, 1Servicio de Medicina Interna, Hospital Virgen de las Nieves, 2Servicio de Medicina Interna, Hospital Clínico San Cecilio, Granada, 3Servicio de Medicina Interna, Hospital Carlos-Haya, Malaga, 4Servicio de Reumatología, Hospital Virgen de la Victoria, Málaga and 5Servicio de Inmunología, Hospital Virgen de las Nieves, Granada, Spain

Correspondence to: E. Sánchez, Instituto de Parasitología y Biomedicina ‘López-Neyra’, CSIC, Parque Tecnológico de Ciencias de la Salud., Avenida del Conocimiento s/n 18100-Armilla (Granada), Spain. E-mail: elena@ipb.csic.es

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SIR, Systemic lupus erythematosus (SLE) is a prototypic autoimmune disease with a complex pathogenesis which involves multiple genetic and environmental factors. Although the pathogenesis of SLE is unknown, a strong genetic component is well established. Among the genetic factors believed to influence susceptibility to SLE, the major histocompatibility complex (MHC) alleles show the most significant association. The mechanism by which these alleles increase the risk of SLE is unknown. However, it seems that regulatory factors of expression of MHC class II molecules could . . . [Full Text of this Article]


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