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Rheumatology Advance Access originally published online on February 5, 2008
Rheumatology 2008 47(4):547-548; doi:10.1093/rheumatology/kem382
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© The Author 2008. Published by Oxford University Press on behalf of the British Society for Rheumatology. All rights reserved. For Permissions, please email: journals.permissions@oxfordjournals.org

LETTERS TO THE EDITOR

Promoter activity of A/G at position –168 in the type III promoter of MHCIITA gene

H. Okamoto, H. Kaneko, C. Terai and N. Kamatani

Institute of Rheumatology, Tokyo Women's Medical University, Tokyo, Japan.

Correspondence to: H. Okamoto, Institute of Rheumatology, Tokyo Women's Medical University, 10-22 Kawada-cho, Shinjuku, Tokyo 162-0054, Japan. E-mail: hokamoto@ior.twmu.ac.jp

The first 10% of the full text of this article appears below.

SIR, The MHC class II transactivator (MHCIITA) gene was originally identified as a defective gene associated with bare lymphocyte syndrome (BLS) [1]. Transcription of the human MHCIITA gene is controlled by alternative promoters [2]. Promoter III (pIII) is responsible for constitutive CIITA expression in B cells.

The recent paper by Swanberg et . . . [Full Text of this Article]


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