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Rheumatology Advance Access originally published online on July 13, 2009
Rheumatology 2009 48(9):1176-1177; doi:10.1093/rheumatology/kep201
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© The Author 2009. Published by Oxford University Press on behalf of the British Society for Rheumatology. All rights reserved. For Permissions, please email: journals.permissions@oxfordjournals.org

Comment on: Investigation of candidate polymorphisms and disease activity in rheumatoid arthritis patients on methotrexate

Wouter M. Kooloos1, Henk-Jan Guchelaar1, Tom W. J. Huizinga2 and Judith A. M. Wessels1

1Department of Clinical Pharmacy and Toxicology and 2Department of Rheumatology, Leiden University Medical Center, Leiden, The Netherlands

Correspondence to: Judith A. M. Wessels, Department of Clinical Pharmacy and Toxicology, Leiden University Medical Center, PO Box 9600, NL 2300 RC, Leiden, The Netherlands. E-mail: j.a.m.wessels@lumc.nl

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SIR, With great interest we read the article of Lee et al. [1] in which associations between candidate polymorphisms and disease activity in RA patients on MTX were assessed. Hereby, the minor allele of the single nucleotide polymorphism (SNP) within the ATIC gene (rs4673993), which is in linkage disequilibrium (LD) with rs2372536, was associated with low disease activity [28-joint disease activity score (DAS28) <=3.2] in a cohort of patients on MTX monotherapy. Previously, our group found an opposite . . . [Full Text of this Article]


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Y. C. Lee, J. Cui, K. H. Costenbader, N. A. Shadick, M. E. Weinblatt, and E. W. Karlson
Comment on: Investigation of candidate polymorphisms and disease activity in rheumatoid arthritis patients on methotrexate: reply
Rheumatology, September 1, 2009; 48(9): 1177 - 1177.
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